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In this regard, it has been proposed that a viral infection in which selfantigens that generate molecular mimicry with myelin proteins cause a loss of tolerance against it, which results in the destruction of myelin mediated by activated T lymphocytes in white matter of the brain and sometimes extending into the gray matter, resulting in defects in the conduction of nerve impulses that leads to symptoms, depending on the affected site of the brain or spinal cord [1] (Figure 1)

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Further research is required to investigate the possible interactions between the presence of oxidative stress and both traditional risk factors (such as dyslipidemia) and non-traditional risk factors (such as inflammation, vascular calcification, advanced glycation end-products and endothelial dysfunction) for atherosclerosis

Association of Depression and Anxiety Disorders With Autoimmune Thyroiditis A Systematic Review and Meta-analysis