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In any case, the authors were self-aware enough to state that: In all, our preliminary observations support the need for prospective, randomized clinical trials with larger numbers of patients to investigate this approach further far from a proclamation that their study is Gospel, but rather that their entire correspondence is simply them saying: We found a potentially interesting effect in a few T1DM patients

G6PD-Deficiency in T cells Based on what has been presented until this point, one may hypothesize how extrinsic signals from other G6PD-d cells might direct a T cell contribution to ASCVD

This is metabolically significant because visceral adipose tissue is strongly associated with metabolic dysfunction, insulin resistance, cardiovascular disease risk, and systemic inflammation