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Bilirubin causes protein oxidation, lipid peroxidation, decreased cellular glutathione content, increased lactate dehydrogenase levels, and nitric oxide release (via neuronal nitric oxide synthase activation via N-methyl-d aspartate receptor activation)

Proteins that are up-regulated in tumor vessels alter vascular function, and may constitute new targets for therapy as discussed further below

A genetic implication of HGF and MET in the development of brain emerges from the literature, and various alterations in the HGF-MET pathway correlate with features of some NDDs such as ASD, schizophrenia, and non-syndromic hearing loss

All efforts were made to minimize animal suffering and the number of animals used