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TC10 Attenuates Cognitive Decline via GLP-1 (2025) This 2025 Scientific Reports (Nature Publishing Group) study is the first to demonstrate that TC10's cognitive protective effects in mice are GLP-1-dependent distinguishing TC10's mechanism from TC8's ketone-based pathway

However, because the drug reduces food intake, these raw materials for detoxification also become scarce

Unlike GLP-1, GIP can promote glucagon secretion in normal and hypoglycemic states, and inhibit it in hyperglycemia, thus acting as a bifunctional hormone to maintain blood glucose homeostasis

Patients treated with the more aggressive schedule were given CT-996 at an initial dose of 10 mg, which was gradually increased every three to seven days to a peak of 120 mg