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The overexpression of miR-21 in UC-MSCs may play a critical role in the restoration of ovarian function in ZP3 peptide-induced autoimmune POI mice, potentially through inhibition of the phosphatase and tensin homolog /Akt/ forkhead box O3a (PTEN/Akt/FOXO3a) signaling pathway

Results of the current model also align with published clinical trials

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