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Also, BPC 157 attenuated and improved the postponed deleterious outcome (lower brain edema, significantly lower number and size of haemorrhagic traumatic lacerations, significantly lower intensity of subarachnoidal bleeding, significantly less intraventricular haemorrhage shown microscopically, declined fatality) (19)

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In addition, MOR-expressing GABAergic afferents (e.g., from RMTg) interact with midbrain GABAergic interneurons and could lead to MOR antagonist-induced DA release [59]