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Higher protein intake was independently protective against lean mass loss in patients on anti-obesity medications
We provide substantial evidence against an enteroendocrine source of GLP-1 as the mechanism for increased plasma insulin in FATP2-KO mice, including (a) no difference between plasma GLP-1 levels or glucose tolerance in db/db FATP2-KO mice following oral versus i.p
Aging involves modifications in connective tissue and cellular regeneration that manifest as a rough-textured appearance and wrinkles
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