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the impacts of adolescent cannabinoid exposure on striatal anxiety- and depressive-like pathophysiology are prevented by the antioxidant N-acetylcysteine

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The resulting death of pericytes leaves the microvasculature continuously constricted, leading to microcirculatory failure and long-term cerebral hypoperfusion due to the contractile apparatus remaining in rigor upon pericyte death, thus reducing capillary diameter [94, 104]